Skip Navigation


Radiation Protection Dosimetry Advance Access originally published online on January 18, 2005
Radiation Protection Dosimetry 2005 113(2):233-235; doi:10.1093/rpd/nch451
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow All Versions of this Article:
113/2/233    most recent
nch451v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (6)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Nikezic, D.
Right arrow Articles by Yu, K. N.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Nikezic, D.
Right arrow Articles by Yu, K. N.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2005. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oupjournals.org

Scientific Note

Are radon gas measurements adequate for epidemiological studies and case control studies of radon-induced lung cancer?

D. Nikezic1 and K. N. Yu2,*

1 Faculty of Science, University of Kragujevac R. Domanovic 12, 34000 Kragujevac Serbia and Montenegro
2 Department of Physics and Materials Science, City University of Hong Kong, Tat Chee Avenue, Kowloon Tong, Hong Kong

* Corresponding author: peter.yu{at}cityu.edu.hk

Received November 14, 2004, accepted December 11, 2004

The lung dose derived from radon is not attributed to the radon gas itself, but instead to its short-lived progeny. However, in many epidemiological studies as well as in case control studies of the radon risk, the excess number of cancers are related to the radon gas exposure, and not to the radon progeny exposure. A justification for such an approach has resorted to the assumption that there is self-compensation between the radiation doses from the unattached and attached fractions. In the present study, we used the Jacobi model to calculate the radon progeny concentrations in a room by varying the attachment rate and then calculated the resulting lung dose. It was found that self-compensation was not fully realised, and the effective dose can vary by a factor up to ~2 for the same radon gas concentration. In conclusion, the radon gas concentration alone does not provide adequate information on the effective dose.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Radiat Prot DosimetryHome page
D. Nikezic and N. Stevanovic
Room model with three modal distributions of attached 220Rn progeny and dose conversion factor
Radiat Prot Dosimetry, January 1, 2007; 123(1): 95 - 102.
[Abstract] [Full Text] [PDF]


Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.